̽��ֱ�� of Cambridge - Imperial College London

Young adults generally more active after starting work, but sleep less – unless working from home

cjb250 — Tue, 28 Jan 2025 00:01:41 +0000

̽��ֱ��increase in physical activity was mainly seen in those doing semi-routine occupations such as bus driving or hairdressing, and routine occupations such as cleaning or waiting, or technical jobs. There was little change seen among people entering managerial or professional occupations.

People who work from home saw a decrease in levels of physical activity – though their sleep levels did not change when they started work.

Young adulthood – ages 16 to 30 years – is an important time in terms of health. Although we are typically at our peak physical health, it is also a time when many risk factors for long term diseases such as heart disease, type 2 diabetes and cancer begin to develop.

Health guidelines recommend young adults get between seven and nine hours of sleep a night, engage in 150 minutes or more of moderate physical activity per week, and consume at least five portions of fruit and vegetables per day.

Young adulthood is also the time when most people start work, which changes their daily routines and activities, resources such as time and money, and social and physical environments – all of which affect health behaviours and health in later life.

To quantify the impact that starting work has on health-related behaviours, a team led by researchers at the Medical Research Council (MRC) Epidemiology Unit at the ̽��ֱ�� of Cambridge examined repeated data taken over time from more than 3,000 participants in the UK Household Longitudinal Study. All the participants were aged 16–30 years and started work for the first time between 2015 and 2023.

̽��ֱ��results are published today in the International Journal of Behavioral Nutrition and Physical Activity.

Dr Eleanor Winpenny, who was based at the ̽��ֱ�� of Cambridge when she carried out the work, but is now at Imperial College London, said: “We know about physical activity and sleep patterns among young people while they’re at school, but very little about what happens when they start work. Given the impact that work can have on our lives – and the lasting impacts this can have on our health – it’s important to try and understand what happens at this transition.”

̽��ֱ��analysis showed that when people started work, their physical activity increased by an amount equivalent to around 28 min of moderate activity (such as cycling) per day on average – but then decreased each year after starting work by around 7 min per day.

̽��ֱ��biggest increase was among males – up by an equivalent of around 45 min of moderate activity per day compared to an increase of around 16 min for females. People who did not have a university degree also showed a greater increase in physical activity compared to those with a university degree – equivalent to around a 42 min increase of moderate physical activity per day compared to 15 min per day.

Working from home, however, appeared to be associated with an initial decrease in physical activity, equivalent to around 32 min of moderate activity per day.

When young adults started work, the amount of time they slept per night dropped immediately by almost 10 minutes and remained stable at this level over time; however, people without a degree showed a continuing decrease of about 3 minutes of sleep per night each year after starting work, while those with a degree slowly increased back to their pre-work sleep levels.

There was little change in the amount of fruit and vegetables consumed after starting work.

Alena Oxenham, from the MRC Epidemiology Unit, said: “Beginning work can have a profound impact on our lifestyles and on behaviours that might make a difference to our health, if not immediately then later in life.

“Although we found that people tend to do more physical activity when they begin work, which is good news, these are averages, and some people – particularly those who work from home and, to a lesser degree, those with office-based jobs – may do less.

“If we want to stay healthy throughout our lives, we need to remember that keeping active is an important way of helping us achieve this goal. Those working at home might want to consider incorporating physical activity into their day, for example by going for a walk before or after work, or during a lunch break.”

Dr Winpenny added: “Workplaces provide an opportunity to create environments and cultures that support healthier diets, more physical activity and better sleep for young adults. This could result in healthier employees and fewer sick days in the immediate term, but also have long term benefits, helping prevent health issues in later life.”

̽��ֱ��research was funded by the MRC and the National Institute for Health and Care Research.

Reference
Oxenham, AF, et al. New job, new habits? A multilevel interrupted time series analysis of changes in diet, physical activity and sleep among young adults starting work for the first time. International Journal of Behavioral Nutrition and Physical Activity; 28 Jan 2025; DOI: 10.1186/s12966-024-01682-8

When young adults start working, the amount of daily physical activity they do increases sharply, only to fall away again over the next few years, while the amount of sleep they get falls slightly, according to new research led by scientists at the ̽��ֱ�� of Cambridge.

If we want to stay healthy throughout our lives, we need to remember that keeping active is an important way of helping us achieve this goal

Alena Oxenham

Roman Koester

Cyclist in London

̽��ֱ��text in this work is licensed under a Creative Commons Attribution-NonCommercial-ShareAlike 4.0 International License. Images, including our videos, are Copyright © ̽��ֱ�� of Cambridge and licensors/contributors as identified. All rights reserved. We make our image and video content available in a number of ways – on our main website under its Terms and conditions, and on a range of channels including social media that permit your use and sharing of our content under their respective Terms.

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Public Domain

How did the building blocks of life arrive on Earth?

sc604 — Fri, 11 Oct 2024 18:00:00 +0000

Volatiles are elements or compounds that change into vapour at relatively low temperatures. They include the six most common elements found in living organisms, as well as water. ̽��ֱ��zinc found in meteorites has a unique composition, which can be used to identify the sources of Earth’s volatiles.

̽��ֱ��researchers, from the ̽��ֱ�� of Cambridge and Imperial College London, have previously found that Earth’s zinc came from different parts of our Solar System: about half came from beyond Jupiter and half originated closer to Earth.

“One of the most fundamental questions on the origin of life is where the materials we need for life to evolve came from,” said Dr Rayssa Martins from Cambridge’s Department of Earth Sciences. “If we can understand how these materials came to be on Earth, it might give us clues to how life originated here, and how it might emerge elsewhere.”

Planetesimals are the main building blocks of rocky planets, such as Earth. These small bodies are formed through a process called accretion, where particles around a young star start to stick together, and form progressively larger bodies.

But not all planetesimals are made equal. ̽��ֱ��earliest planetesimals that formed in the Solar System were exposed to high levels of radioactivity, which caused them to melt and lose their volatiles. But some planetesimals formed after these sources of radioactivity were mostly extinct, which helped them survive the melting process and preserved more of their volatiles.

In a study published in the journal Science Advances, Martins and her colleagues looked at the different forms of zinc that arrived on Earth from these planetesimals. ̽��ֱ��researchers measured the zinc from a large sample of meteorites originating from different planetesimals and used this data to model how Earth got its zinc, by tracing the entire period of the Earth’s accretion, which took tens of millions of years.

Their results show that while these ‘melted’ planetesimals contributed about 70% of Earth’s overall mass, they only provided around 10% of its zinc.

According to the model, the rest of Earth’s zinc came from materials that didn’t melt and lose their volatile elements. Their findings suggest that unmelted, or ‘primitive’ materials were an essential source of volatiles for Earth.

“We know that the distance between a planet and its star is a determining factor in establishing the necessary conditions for that planet to sustain liquid water on its surface,” said Martins, the study’s lead author. “But our results show there’s no guarantee that planets incorporate the right materials to have enough water and other volatiles in the first place – regardless of their physical state.”

̽��ֱ��ability to trace elements through millions or even billions of years of evolution could be a vital tool in the search for life elsewhere, such as on Mars, or on planets outside our Solar System.

“Similar conditions and processes are also likely in other young planetary systems,” said Martins. “ ̽��ֱ��roles these different materials play in supplying volatiles is something we should keep in mind when looking for habitable planets elsewhere.”

̽��ֱ��research was supported in part by Imperial College London, the European Research Council, and UK Research and Innovation (UKRI).

Reference:
Rayssa Martins et al. ‘Primitive asteroids as a major source of terrestrial volatiles.’ Science Advances (2024). DOI: 10.1126/sciadv.ado4121

Researchers have used the chemical fingerprints of zinc contained in meteorites to determine the origin of volatile elements on Earth. ̽��ֱ��results suggest that without ‘unmelted’ asteroids, there may not have been enough of these compounds on Earth for life to emerge.

Rayssa Martins/Ross Findlay

An iron meteorite from the core of a melted planetesimal (left) and a chondrite meteorite, derived from a ‘primitive’, unmelted planetesimal (right).

Yes

Genetic variant linked to lower levels of HIV virus in people of African ancestry

cjb250 — Wed, 02 Aug 2023 15:00:57 +0000

Reported today in Nature, this is the first new genetic variant related to HIV infection discovered in over 25 years of research. It could, in the future, help direct the development of new treatment approaches for those living with HIV.

HIV remains a major threat to global health. According to UNAIDS, there were 38.4 million people living with HIV globally in 2021. A combination of pre-exposure drugs and medicines that dramatically reduce viral loads has had a major impact on transmission, yet 1.5 million people were newly infected in 2021. And while treatments have improved dramatically since the virus was first identified, 650,000 people still died from AIDS-related illnesses in that year.

Viral load is the amount of a virus in a patient’s system. Higher levels are known to correlate with faster disease progression and increased risk of transmission. But viral load varies widely among infected individuals, influenced by a number of factors including an individual’s genetic makeup.

Most of what we know about the relationship between our DNA and HIV comes from studies among European populations. But given that HIV disproportionately affects people on the African continent – more than 25 million people who are HIV-positive live on the continent – it’s important to better understand the role of genetics in HIV infection in African populations.

To investigate this question, researchers analysed the DNA of almost 4,000 people of African ancestry living with HIV-1, the most common type of the virus. They identified a variant within a region on chromosome 1 containing the gene CHD1L which was associated with reduced viral load in carriers of the variant. Between 4% and 13 % of people of African origin are thought to carry this particular variant.

Paul McLaren from the Public Health Agency of Canada’s National Microbiology Laboratory, joint first author, said: “African populations are still drastically underrepresented in human DNA studies, despite experiencing the highest burden of HIV infection. By studying a large sample of people of African ancestry, we’ve been able to identify a new genetic variant that only exists in this population and which is linked to lower HIV viral loads.”

CHD1L is known to play a role in repairing damaged DNA, though it is not clear why the variant should be important in reducing viral load. However, as HIV attacks immune cells, researchers at the ̽��ֱ�� of Cambridge’s Department of Medicine, led by Dr Harriet Groom and Professor Andrew Lever, used stem cells to generate variants of cells that HIV can infect in which CHD1L had either been switched off or its activity turned down.

HIV turned out to replicate better in a type of immune cell known as a macrophage when CHD1L was switched off. In another cell type, the T cell, there was no effect – perhaps surprising since most HIV replication occurs in the latter cell type.

Dr Groom said: “This gene seems to be important to controlling viral load in people of African ancestry. Although we don’t yet know how it’s doing this, every time we discover something new about HIV control, we learn something new about the virus and something new about the cell. ̽��ֱ��link between HIV replication in macrophages and viral load is particularly interesting and unexpected.”

Co-author Professor Manjinder Sandhu from the Faculty of Medicine at Imperial College London said: “With more than a million new HIV infections a year, it’s clear that we still have a long way to go in the fight against HIV – we are yet to have a vaccine to prevent infection, have yet to find a cure and still see drug resistance emerging in some individuals. ̽��ֱ��next step is to fully understand exactly how this genetic variant controls HIV replication.”

̽��ֱ��research in Cambridge was largely funded by the Medical Research Council. A full list of funders can be found in the research paper.

Dr Groom is a Research Fellow at Sidney Sussex College and Professor Andrew Lever is a Professorial Fellow Peterhouse, Cambridge.

Reference
McLaren, PJ; Porreca, I; Iaconis, G; Mok, HP, Mukhopadhyay, Sl; Karakoc, E et al. Africa-specific human genetic variation near CHD1L associates with HIV-1 load. Nature; 2 Aug 2023; DOI: 10.1038/s41586-023-06370-4

An international team of researchers has found a genetic variant that may explain why some people of African ancestry have naturally lower viral loads of HIV, reducing their risk of transmitting the virus and slowing progress of their own illness.

Every time we discover something new about HIV control, we learn something new about the virus and something new about the cell

Harriet Groom

jonrawlinson

Know your HIV status sign in Simonga village, Zambia

̽��ֱ��text in this work is licensed under a Creative Commons Attribution-NonCommercial-ShareAlike 4.0 International License. Images, including our videos, are Copyright © ̽��ֱ�� of Cambridge and licensors/contributors as identified. All rights reserved. We make our image and video content available in a number of ways – as here, on our main website under its Terms and conditions, and on a range of channels including social media that permit your use and sharing of our content under their respective Terms.

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Licence type:

Attribution

Listening from afar

jg533 — Wed, 14 Sep 2022 10:50:20 +0000

A new, hands-off approach to monitoring biodiversity is saving time and money, and helping to identify sites where intervention is most needed.

Cognitive impairment from severe COVID-19 equivalent to 20 years of ageing, study finds

cjb250 — Tue, 03 May 2022 07:00:18 +0000

̽��ֱ��findings, published in the journal eClinicalMedicine, emerge from the NIHR COVID-19 BioResource. ̽��ֱ��results of the study suggest the effects are still detectable more than six months after the acute illness, and that any recovery is at best gradual.

There is growing evidence that COVID-19 can cause lasting cognitive and mental health problems, with recovered patients reporting symptoms including fatigue, ‘brain fog’, problems recalling words, sleep disturbances, anxiety and even post-traumatic stress disorder (PTSD) months after infection. In the UK, a study found that around one in seven individuals surveyed reported having symptoms that included cognitive difficulties 12 weeks after a positive COVID-19 test.

While even mild cases can lead to persistent cognitive symptoms, between a third and three-quarters of hospitalised patients report still suffering cognitive symptoms three to six months later.

To explore this link in greater detail, researchers analysed data from 46 individuals who received in-hospital care, on the ward or intensive care unit, for COVID-19 at Addenbrooke’s Hospital, part of Cambridge ̽��ֱ�� Hospitals NHS Foundation Trust. 16 patients were put on mechanical ventilation during their stay in hospital. All the patients were admitted between March and July 2020 and were recruited to the NIHR COVID-19 BioResource.

̽��ֱ��individuals underwent detailed computerised cognitive tests an average of six months after their acute illness using the Cognitron platform, which measures different aspects of mental faculties such as memory, attention and reasoning. Scales measuring anxiety, depression and post-traumatic stress disorder were also assessed. Their data were compared against matched controls.

This is the first time that such rigorous assessment and comparison has been carried out in relation to the after effects of severe COVID-19.

COVID-19 survivors were less accurate and with slower response times than the matched control population – and these deficits were still detectable when the patients were following up six months later. ̽��ֱ��effects were strongest for those who required mechanical ventilation. By comparing the patients to 66,008 members of the general public, the researchers estimate that the magnitude of cognitive loss is similar on average to that sustained with 20 years ageing, between 50 and 70 years of age, and that this is equivalent to losing 10 IQ points.

Survivors scored particularly poorly on tasks such as verbal analogical reasoning, a finding that supports the commonly-reported problem of difficulty finding words. They also showed slower processing speeds, which aligns with previous observations post COVID-19 of decreased brain glucose consumption within the frontoparietal network of the brain, responsible for attention, complex problem-solving and working memory, among other functions.

Professor David Menon from the Division of Anaesthesia at the ̽��ֱ�� of Cambridge, the study’s senior author, said: “Cognitive impairment is common to a wide range of neurological disorders, including dementia, and even routine ageing, but the patterns we saw – the cognitive 'fingerprint' of COVID-19 – was distinct from all of these.”

While it is now well established that people who have recovered from severe COVID-19 illness can have a broad spectrum of symptoms of poor mental health – depression, anxiety, post-traumatic stress, low motivation, fatigue, low mood, and disturbed sleep – the team found that acute illness severity was better at predicting the cognitive deficits.

̽��ֱ��patients’ scores and reaction times began to improve over time, but the researchers say that any recovery in cognitive faculties was at best gradual and likely to be influenced by a number of factors including illness severity and its neurological or psychological impacts.

Professor Menon added: “We followed some patients up as late as ten months after their acute infection, so were able to see a very slow improvement. While this was not statistically significant, it is at least heading in the right direction, but it is very possible that some of these individuals will never fully recover.”

There are several factors that could cause the cognitive deficits, say the researchers. Direct viral infection is possible, but unlikely to be a major cause; instead, it is more likely that a combination of factors contribute, including inadequate oxygen or blood supply to the brain, blockage of large or small blood vessels due to clotting, and microscopic bleeds. However, emerging evidence suggests that the most important mechanism may be damage caused by the body’s own inflammatory response and immune system.

While this study looked at hospitalised cases, the team say that even those patients not sick enough to be admitted may also have tell-tale signs of mild impairment.

Professor Adam Hampshire from the Department of Brain Sciences at Imperial College London, the study’s first author, said: “Around 40,000 people have been through intensive care with COVID-19 in England alone and many more will have been very sick, but not admitted to hospital. This means there is a large number of people out there still experiencing problems with cognition many months later. We urgently need to look at what can be done to help these people.”

Professor Menon and Professor Ed Bullmore from Cambridge’s Department of Psychiatry are co-leading working groups as part of the COVID-19 Clinical Neuroscience Study (COVID-CNS) that aim to identify biomarkers that relate to neurological impairments as a result of COVID-19, and the neuroimaging changes that are associated with these.

̽��ֱ��research was funded by the NIHR BioResource, NIHR Cambridge Biomedical Research Centre and the Addenbrooke’s Charitable Trust, with support from the NIHR Cambridge Clinical Research Facility.

Reference
Hampshire, A et al. Multivariate profile and acute-phase correlates of cognitive deficits in a COVID-19 hospitalised cohort. eClinicalMedicine; 28 Apr 2022; DOI: 10.1016/j.eclinm.2022.101417

Cognitive impairment as a result of severe COVID-19 is similar to that sustained between 50 and 70 years of age and is the equivalent to losing 10 IQ points, say a team of scientists from the ̽��ֱ�� of Cambridge and Imperial College London.

Cognitive impairment is common to a wide range of neurological disorders, but the patterns we saw – the cognitive 'fingerprint' of COVID-19 – was distinct from all of these

David Menon

RUBEN BONILLA GONZALO

Senior woman wearing face mask lying on hospital bed

̽��ֱ��text in this work is licensed under a Creative Commons Attribution 4.0 International License. Images, including our videos, are Copyright © ̽��ֱ�� of Cambridge and licensors/contributors as identified. All rights reserved. We make our image and video content available in a number of ways – as here, on our main website under its Terms and conditions, and on a range of channels including social media that permit your use and sharing of our content under their respective Terms.

Yes

Curbing COVID-19 in schools: Cambridge scientists support CO2 monitor rollout

sc604 — Mon, 17 Jan 2022 12:44:06 +0000

Scientists from Cambridge, Surrey and Imperial College London are supporting the rollout of portable monitors to UK schools as part of project CO-TRACE. ̽��ֱ��researchers behind the collaboration have produced materials to help teachers use the monitors, which have been rolled out to schools nationwide.

̽��ֱ��level of carbon dioxide (CO₂) in a closed space is a good indicator of air quality and can signpost the need for ventilation. As the virus that causes COVID-19 is airborne, ensuring the air is properly refreshed using ventilation is crucial for reducing its spread. ̽��ֱ��device displays levels of CO2 and colour coding to indicate good, normal, or poor ventilation. Well ventilated spaces should have CO2 levels consistently below 800 parts per million (ppm), with readings above 1500ppm indicating poor ventilation or overcrowding.

“CO2 monitors allow teachers to assess the ventilation in their classrooms for the first time,” said Imperial’s Dr Henry Burridge, co-investigator on the project. “This is especially important during colder months when ventilation is typically lower due to colder outdoor temperatures, causing COVID-19 and other airborne diseases like the common cold and flu to linger and spread more easily.”

̽��ֱ��monitors mean teachers can see CO2 levels change in real-time as windows are opened and air is refreshed, allowing them to balance ventilation and warmth. Teachers can also use the monitors to know when it is safe to close windows slightly, which could help them keep classrooms more comfortable. As well as being a good ‘proxy’ for ventilation, lower CO2 levels have been linked to improved learning outcomes and better cognitive performance.

̽��ֱ��team behind the CO-TRACE project uses experimental modelling, numerical simulations, full-scale observations, and infection risk modelling to understand how the potential for COVID-19 spread changes with indoor air flows, ventilation levels, and the number of people in a space. In 2021, the researchers used monitored CO2 to indicate how much exhaled breath was present within classrooms, and their models found that seasonal variation in classroom ventilation levels could lead to airborne infection risks in winter being roughly double those in summer. This highlights that monitoring excess CO2 could be of significant benefit in mitigating airborne infection risk.

̽��ֱ��portability of the CO2 monitors, supplied by the Department for Education (DfE), means schools can move them around to test different areas, starting with those they suspect may be poorly ventilated.

“ ̽��ֱ��monitors empower teachers to strike a balance between good ventilation and warmth during winter,” said Professor Paul Linden from Cambridge’s Department of Applied Mathematics and Theoretical Physics, co-investigator on the programme. “We are pleased that the Government is taking evidence-based action to address air quality and COVID-19 spread in schools.”

̽��ֱ��monitors are accompanied by advice from the project which guides appropriate actions from teacher based on the CO2 readings in classrooms. Recommendations include opening higher windows before lower ones, and closing windows slowly when ventilation is good.

Schools with areas that are consistently low in air quality despite ventilation should consider using air cleaners. For such schools, the DfE is distributing between 7,000 and 8,000 air cleaning units.

When the project was announced in 2021, then-Education Secretary Gavin Williamson said: “Providing all schools with CO2 monitors will help them make sure they have the right balance of measures in place, minimising any potential disruption to education and allowing them to focus on world-class lessons and catch up for the children who need it. By keeping up simple measures such as ventilation and testing, young people can now enjoy more freedom at school and college.”

̽��ֱ��project is funded by the EPSRC, part of UK Research and Innovation (UKRI).

Adapted from an Imperial College story.

UK schools have received more than 300,000 CO2 monitors as part of a government initiative to reduce COVID-19 spread in classrooms.

̽��ֱ��monitors empower teachers to strike a balance between good ventilation and warmth during winter

Paul Linden

Olivier Le Moal

CO2 monitor

Yes

̽��ֱ��Internet of Stings: research will probe privacy and legal concerns of smart devices

sc604 — Fri, 22 Oct 2021 08:43:11 +0000

These questions have been worrying researchers at the ̽��ֱ�� of Cambridge Department of Computer Science and Technology. Now they are launching a year-long investigation into the ways our information is being collected and whether or not these always comply with regulations and the law.

Working in collaboration with colleagues at Imperial College London, they will probe the data that flows from the Internet of Things – the networked consumer devices, such as smart printers, doorbells and toys, that are an increasing presence in our homes.

Backed by a grant from the Information Commissioner’s Office, the UK’s data protection regulator, they will be investigating what Dr Jat Singh describes as ‘the Internet of Stings’.

Research shows that information from our devices often finds its way to a range of third parties, such as user-tracking and advertising networks that may mine it for valuable information about consumer behaviour. He’s also worried about the occasions when data is transmitted from one country to another where there may be different rules, rights and restrictions around data and its use.

So Singh and the research team want to investigate the transmission of data from our devices to find out if it is in line with relevant law – and to inform consumers about the potential of what we can do to have better control over our information.

“We see ‘smart’ devices increasingly being worn on people's bodies and used in people's homes,” said Singh. “However, it’s often unclear what happens with the data these devices collect: where that data goes and how it is used. This is concerning, given these devices can often collect highly personal, private and sensitive information about ourselves and our lives.

“This project seeks to shed light on the state of current commercial data practices by analysing the nature of data flows from both a technical and a data rights, perspective. We aim to show if there are any data protection implications and concerns in the consumer smart device landscape so that we can empower policymakers, regulators, and individuals alike.”

Dr Singh leads the Compliant & Accountable Systems Research group, a team of researchers working at the intersection of technology and law. They consider ways in which technology could be better designed and deployed to meet legal and regulatory concerns and work to inform policymakers and regulators about the technical realities of new and emerging technologies.

Technical network-monitoring mechanisms have been used to establish the ways in which data is transmitted, the patterns of transmissions, and the destinations it ended up in. “This showed that potentially problematic data-flow appears to be rife in the Internet of Things,” said Singh.

Over the next year, they’ll be taking a detailed look at whether devices actually transmit data in accordance with the privacy policies and other legal obligations of the companies that sell them.

They will also explore the implications of mitigations that consumers might use, such as the implications of blocking particular data flows.

They want to establish the nature and scale of any problems and see if vendor companies are being honest and fully transparent with their consumers and compliant with data protection and other laws. They also want to better inform not only device owners but also regulators and policy-makers about the suspected issues, which may help inform future interventions.

“Problems with the data practices of the consumer smart devices have been suspected for some time, but not fully examined – from both a technical and legal perspective,” said Singh. “We need to do so if we want a better, fairer and more compliant Internet of Things.”

Originally published on the Computer Science and Technology website.

What happens to all the sensitive personal information our smart devices collect from us? Where does the data picked up by our smart watches, speakers and TVs go, who has access to it and how is it used?

It’s often unclear what happens with the data these devices collect: where that data goes and how it is used. This is concerning, given these devices can often collect highly personal, private and sensitive information about ourselves and our lives

Jat Singh

Howard Bouchevereau via Unsplash

Smart speaker

Yes

Scientists develop model to assess COVID-19 infection risk in offices and schools

sc604 — Wed, 06 Oct 2021 10:56:27 +0000

̽��ֱ��model – developed by researchers at the ̽��ֱ�� of Cambridge, Imperial College London and the ̽��ֱ�� of Leeds – uses monitored CO2 and occupancy data to predict how many workers are likely to be infected by an asymptomatic but infectious colleague.

Applications of the infection model have demonstrated that most workers in well-ventilated, quiet offices are unlikely to infect each other via airborne particles, but the risk becomes greater if the space is poorly ventilated or if the workers are involved in activities that require more speaking. For instance, the model predicts each infected person could infect two to four others in an adequately ventilated but noisy call centre. Risks are also likely to increase if the infected individual is a ‘super spreader’.

̽��ֱ��model also suggests that halving the occupancy of an office could reduce the risk of airborne transmission four-fold. ̽��ֱ��results are reported in the journal Indoor and Built Environment.

In areas with lower ventilation rates and high occupancy, CO2 levels are higher, so monitoring them can provide a warning to building managers to identify areas where the risk of airborne transmission of COVID-19 are higher. Achievable interventions can then be made, for instance, to improve ventilation or change worker attendance patterns to reduce occupancy.

In shared spaces such as offices and classrooms, exposure to infectious airborne matter builds up, and room occupancy may vary. By using carbon dioxide levels as a proxy for exhaled breath, the model can assess the variable exposure risk as people come and go.

“Ventilation is complicated and airflow is invisible, so it’s hard for people to appreciate the effects in the home or workplace,” said co-author Professor Paul Linden from Cambridge’s Department of Applied Mathematics and Theoretical Physics. “Commercially available CO2 monitors are being installed in schools and I would recommend their installation in the workplace.”

“Our work emphasises the importance of good ventilation in workplaces and in schools,” said lead author Dr Henry Burridge, from Imperial College London. “ ̽��ֱ��model demonstrates that by managing the ventilation and occupancy levels of shared spaces we can manage the risk of airborne infection by a virus such as that which causes COVID-19.”

“ ̽��ֱ��appropriate use of tools such as CO2 monitoring can give building managers a much better understanding of their own ventilation systems and how they are performing for each activity undertaken in the space,” said Professor Andrew Curran, Chief Scientific Adviser at the Health and Safety Executive and lead for the PROTECT study. “For most businesses, a COVID-19 control strategy will involve a blended combination of measures identified through a risk assessment – there is no silver bullet.”

̽��ֱ��research was funded by the PROTECT COVID-19 National Core Study and the Engineering and Physical Sciences Research Council (EPSRC, part of UK Research and Innovation).

Reference:
Henry C Burridge et al. ‘Predictive and retrospective modelling of airborne infection risk using monitored carbon dioxide.’ Indoor and Built Environment (2021). DOI: 10.1177/1420326X211043564

Adapted from an HSE press release.

As more UK workers and students return to offices and schools, a new model has been developed to predict the risk of airborne COVID-19 infection in such environments.

Ventilation is complicated and air flow is invisible, so it’s hard for people to appreciate the effects in the home or workplace

Paul Linden

Israel Amdrade via Unsplash

People in office sitting in front of computers

Yes